论文部分内容阅读
本文简介近二十年关于炎症与抗炎理论研究的进展。在机体受损伤时所出现的炎症是以局部反应为主的全身性反应,是机体的防御反应之一,可以将炎症分为血管通透性亢进、白细胞游走和增殖等三期。实验性炎症模型也有了不少较为深入的研究。引起炎症的主要化学介质有组织胺、5-羟色胺、激肽、激活的补体系统、溶酶体和前列腺素等。目前认为抗炎药物的作用原理是:甾体类稳定溶酶体膜,非甾体类抑制前列腺素的合成。许多方法可用于抗炎中药的研究。
This article describes nearly 20 years of research on the theory of inflammation and anti-inflammatory progress. Inflammation occurs when the body is damaged, which is a systemic reaction characterized mainly by local reactions. It is one of the body’s defensive responses and can be classified into three stages of hypervascular vascular permeability, leukocyte migration and proliferation. Experimental inflammatory model also has a lot of more in-depth study. The main chemical mediators of inflammation are histamine, serotonin, kinins, activated complement system, lysosomes and prostaglandins. Now that the principle of anti-inflammatory drugs are: steroid stable lysosomal membrane, non-steroidal inhibit prostaglandin synthesis. Many methods can be used for anti-inflammatory Chinese medicine research.