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对于在糖尿病人群中使高血压产生和持续的基本机制,人们认识尚浅。肥胖、胰岛素、遗传因素和钙的体内平衡异常都可能与Na~+/H~+的对输活性增加有关。在第一部分研究中,Na~+/Li~+反向转运活性增加且有高血压的胰岛素依赖性糖尿病(IDDM)患者同Na~+/Li~+反向转运活性正常而有高血压的IDDM患者或血压正常的IDDM患者相比,其全身葡萄糖利用能力低下,胰岛素激活前臂碳水化合物氧化的作用下降,超声检查肾
There is still little understanding of the underlying mechanisms underlying the development and persistence of hypertension in people with diabetes. Obesity, insulin, genetic factors and calcium homeostasis may be related to Na ~ + / H ~ + on the increase of the transmission activity. In the first part of the study, IDDM patients with elevated Na ~ + / Li ~ + antiporter activity and hypertensive insulin-dependent diabetes mellitus (IDDM) with normal Na ~ + / Li ~ + translocation activity and high blood pressure In patients with normotensive IDDM patients, their systemic glucose utilization is low, the effect of insulin-activated forearm carbohydrate oxidation is reduced, and ultrasound is used to examine the kidney