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分娩的发动与炎症因子有关。热休克蛋白70,既是分子伴侣,又是炎症因子——白细胞介素6(IL-6)、白细胞介素1β(IL-1β)的强力诱导剂和强效的免疫佐剂。研究证明,心肌组织中热休克蛋白27表达上调可导致心肌的收缩功能增强。为探讨子宫平滑肌组织中热休克蛋白70在临产前后的表达变化及其与分娩发动的关系,我们进行了研究,现将结果报道如下。一、资料与方法1.标本来源与分组:选择2000年3月—2003年12月中南大学湘雅医院住院分娩、并同意参与研究的30例单胎初产妇,孕周38～41周,均无妊娠合并症,因胎位异常或胎儿窘迫,在硬膜外麻醉下行择期或产时剖宫产分娩。根据临产
The onset of childbirth and inflammatory factors. Heat shock protein 70 is both a chaperone and a potent immune adjuvant as well as a potent inducer of interleukin 6 (IL-6), interleukin 1 beta (IL-1 beta). Studies have shown that the upregulation of heat shock protein 27 in myocardial tissue can lead to enhanced myocardial contractile function. To investigate the expression of heat shock protein 70 in uterine smooth muscle tissue before and after labor changes and its relationship with labor onset, we conducted a study, the results reported below. Materials and methods 1. Specimen source and grouping: March 2000 - December 2003 Xiangya Hospital of Central South University hospital delivery, and agreed to participate in the study of 30 cases of single mothers, gestational age 38 to 41 weeks, were No pregnancy complications, due to fetal abnormalities or fetal distress, elective epidural anesthesia or cesarean delivery. According to labor