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本文测定了103例≥36周的非糖尿病婴儿的羊水和脐血中的C-肽胰岛素和葡萄糖水平,用以观察胎儿宫内生长情况。围产期发病率和死亡率的增高起因于胎儿宫内生长迟缓(IUGR)。IUGR常被认为发生于妊娠末3个月的早期,可能是由于母体的各种情况降低了子宫胎盘血流量引起。胰岛素能促进细胞对氨基酸的吸收,继之合成蛋白质,因此胰岛素被认为是调节胎儿宫内生长的关键,另外,胰岛素促进糖原和脂类沉积于心、肝、肌肉和脂肪组织。现已公认,通过胎盘母血糖转给胎儿的血糖平均水平是胎
We measured the levels of C-peptide insulin and glucose in amniotic fluid and cord blood from 103 non-diabetic infants ≥36 weeks to observe intrauterine growth. Perinatal morbidity and mortality increase due to fetal intrauterine growth retardation (IUGR). IUGR is often thought to occur early in the third trimester of pregnancy and may be due to reduced maternal placental blood flow due to maternal conditions. Insulin can promote the absorption of amino acids, followed by the synthesis of protein, so insulin is considered to be the key to regulating intrauterine growth. In addition, insulin promotes the deposition of glycogen and lipids in heart, liver, muscle and adipose tissue. It is now recognized that the average level of blood glucose transferred to the fetus by placental maternal glucose is the fetus