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肺炎衣原体具有感染动脉的能力和途径 ;体外细胞培养和动物实验证实 :在动脉粥样硬化形成的各阶段 ,肺炎衣原体与靶细胞相互作用 ,促进动脉粥样硬化的发生与发展 ,加速其进程 ;肺炎衣原体感染的兔模型能够发生动脉粥样硬化的病理变化 ;抗生素的干预具有抑制动脉粥样硬化形成的作用 ;基因多态性与基因突变影响个体对肺炎衣原体的易感性。
Chlamydia pneumoniae has the ability to infect arteries and pathways; cell culture in vitro and animal experiments confirmed that: in the formation of atherosclerotic stages of Chlamydia pneumoniae and target cells interact to promote the occurrence and development of atherosclerosis to speed up its process; Chlamydia pneumoniae in rabbits infected with pathological changes of atherosclerosis; antibiotic intervention has the role of inhibiting the formation of atherosclerosis; genetic polymorphisms and gene mutations affect the individual susceptibility to Chlamydia pneumoniae.