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自1935年Stein及Leventhal氏报导后,许多学者提出多囊卵巢病(PCO)的有关病因理论,但多缺乏事实根据。近年来,发展的新技术,使本病内分泌情况更清楚。本文复习激素上的新发现来推想病因。一、症状学和组织学 1962年Goldzieher等复习PC02,200例文献,提出三组基本症状:(一)证实为慢性无排卵,有闭经、不孕、功能性或无排卵性出血症状。(二)有雄激素过多、多毛、男性化。(三)合成代谢增加。半数病人开始肥胖。检查卵巢增大,包囊厚,有不同期的囊性滤泡或萎缩白体,无近期排卵。滤泡周围细胞常有黄体化。但无独特的组织学特
Since Stein and Leventhal’s report in 1935, many scholars have put forward the theory of etiology of polycystic ovarian disease (PCO), but many lack of factual basis. In recent years, the development of new technologies to make this disease more clearly the situation of endocrine. This article reviews the new findings on hormones to infer the etiology. First, the symptoms and histology 1962 Goldzieher et al reviewed PC02, 200 cases of literature, made three basic symptoms: (A) confirmed as chronic anovulation, amenorrhea, infertility, functional or anovulatory bleeding symptoms. (B) Androgen excess, hirsutism, masculine. (C) anabolic increased. Half of the patients started obesity. Check ovarian enlargement, cyst thickness, there are different stages of cystic follicles or atrophic white body, no recent ovulation. Follicles around the cells often luteinized. But no unique histological special