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静脉内溶栓改善了急性心肌梗死(AMI)病人的左室功能和存活,相应降低了病人的危险性,即出现一批低危病人。但溶栓后并没有消除血栓形成对血管的刺激,远处存活心肌仍由一支存在着持续活动性损伤的血管供血,这种情况大多以散在的坏死斑为标志。应用链激酶和组织型纤搭酶元激活剂后,血管再闭塞的发生和反复心肌缺血的倾向,可能是不稳定血管内皮损伤的反应。意大利多中心“残留局部缺血”研究评价了溶栓后早期反复心肌缺血的发生率及预后意义。方法选择无AMI既往史、病发后6h内入冠
Intravenous thrombolysis improves left ventricular function and survival in patients with acute myocardial infarction (AMI), correspondingly reducing the patient’s risk of developing a number of low-risk patients. However, after thrombolysis did not eliminate the blood vessels to stimulate thrombosis, the survival of distant myocardium is still a sustained activity of the existence of vascular blood vessels, mostly in the form of scattered necrotic markers. After the application of streptokinase and tissue-type phospholipase activator, the occurrence of recanalization and the tendency of repeated myocardial ischemia may be the reaction of unstable vascular endothelial injury. The multicenter Italian “Residual Ischemia” study evaluated the incidence of early post-thrombolysis repeated myocardial ischemia and its prognostic significance. Methods None AMI past history, 6h after onset into the crown