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目的 探讨高压氧对脑梗塞急性期病人血浆 α颗粒膜蛋白 ( GMP-1 4 0 )含量变化的机理。方法 应用双夹心放免测定法 ,定量测定两组脑梗塞急性期病人治疗前和治疗 2 0天后的血浆 GMP-1 4 0含量。结果 1脑梗塞急性期患者治疗前的血浆 GMP-1 4 0明显增高 ,与正常值相比有非常显著性差异 ;2治疗 2 0天后 ,高压氧组与对照组相比 ,血浆 GMP-1 4 0含量明显下降 ,两组相比有显著性差异。结论 对于脑梗塞急性期患者 ,在进行常规药物治疗同时 ,加用高压氧治疗 ,可明显降低血小板的活性。
Objective To investigate the mechanism of hyperbaric oxygen on the plasma level of α-granule membrane protein (GMP-1 40) in patients with acute cerebral infarction. Methods Double-Gated Radioimmunoassay (RIA) was used to quantitatively determine plasma GMP-140 levels in patients with acute cerebral infarction before treatment and after treatment for 20 days. Results Acute cerebral infarction patients before treatment plasma GMP-1 40 was significantly higher than the normal value of a very significant difference; 2 treatment 20 days, hyperbaric oxygen group compared with the control group, plasma GMP-1 4 0 content decreased significantly compared with the two groups had significant differences. Conclusions For patients with acute cerebral infarction, platelet activity may be significantly reduced while conventional treatment with hyperbaric oxygen therapy.