论文部分内容阅读
例1,男,3岁。4h 前食(?)已变酸的甘蔗50cm,食后即呕吐4次,腹痛,阵发性抽搐,二便失禁,急诊入院。查体:BP9.3/5.3kPa,呈昏迷状,呼吸急促,脉搏细速,双瞳孔等大等圆,光反应迟钝。颈抗、心肺未见著变。四肢呈痉挛性瘫,腹壁反射消失,克氏、布氏及巴氏征均阳性。入院后经吸氧、扩容、纠酸、止痉及降颅压等治疗,患儿血压复常,压眶上神经有反应。次日仍间断抽搐,四肢呈去大脑强直状态。再配合针灸等治疗,但言语仍障碍,肢体仍瘫,成为后遗症。经随访得知,后因继发感染死亡。
Example 1, male, 3 years old. 4h before eating (?) Sour sugar cane 50cm, vomiting after eating 4 times, abdominal pain, paroxysmal convulsions, second incontinence, emergency admission. Physical examination: BP9.3 / 5.3kPa, was coma, shortness of breath, pulse fine speed, double pupil and other large circle, light reaction slow. Cervical anti-cardiopulmonary no change. Extremities were spastic paralysis, abdominal reflex disappeared, Kirschner, Brinell and Pakistan’s signs were positive. After admission by oxygen, expansion, correcting acid, antispasmodic and intracranial pressure and other treatment, children with normotension, supraorbital nerve pressure response. The next day still intermittent convulsions, limbs were going to the brain ankylosis. Combined with acupuncture and other treatment, but speech is still obstacles, limbs are still paralyzed, become a sequelae. After follow-up that after death due to secondary infection.