通过对75例有机磷中毒迟发性神经病的临床分析,发现此病发生率较高,且主要见于重度中毒病人,其发生与胆碱酯酶活性受抑制无关而与神经靶点酯酶活性受抑制有关,以周围神经损害为主要表现,可产生神经性肌萎缩,经积极治疗绝大多数可以痊愈,呈可逆性与康复缓慢为特点的病理过程,强调在抢救急性有机磷中毒的同时要预防迟发性神经病的出现,而一旦发生要及早发现并积极治疗。 Through the clinical analysis of 75 cases of organophosphate poisoning delayed neuropathy, found that the incidence of this disease is higher, and mainly seen in patients with severe poisoning, its occurrence has nothing to do with the inhibition of cholinesterase activity and nerve target esterase activity by Inhibition of peripheral nerve damage as the main performance can produce neuromuscular atrophy, the vast majority of active treatment can be cured, was reversible and slow recovery characterized pathological process, emphasizing the rescue of acute organophosphate poisoning at the same time to prevent The occurrence of delayed neuropathy, and in the event of an early detection and active treatment.