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C_3e是C_3降解晚期的活性片段,电泳迁移率位于前白蛋白,其分子量为10,000~12,000.给家兔静脉注射C_3e能引起血液中白细胞增多,皮内注射时可增加血管的通透性并有细胞渗出.人的多形核白细胞(PMNS)上有C_3e受体,本文结果表明C_3e与PMNS受体相互作用导致溶酶体酶的释放,某些细胞能从人的C_5中产生趋化活性.将细胞松解素-B(5μg/ml)处理的PMNS加入不同浓度的C_3e中,37℃孵育60分钟,然后测定上清液中β-葡萄糖苷酶、
C_3e is an active fragment of late C_3 degradation, and the electrophoretic mobility is located in prealbumin with a molecular weight of 10,000 to 12,000. Intravenous injection of C_3e to rabbits can cause leukocytosis in the blood and increase vascular permeability when injected intradermally Cell exudation. Human polymorphonuclear leukocytes (PMNS) have C_3e receptors, the results show that C_3e and PMNS receptors lead to the release of lysosomal enzymes, some cells can produce chemotactic activity from human C_5 PMNS treated with cytokinin-B (5μg / ml) were added to different concentrations of C_3e, incubated at 37 ℃ for 60 minutes, then the supernatant was assayed for β-glucosidase,