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以往研究认为亚急性甲状腺炎(De Quervain氏甲状腺炎)之主要特征为甲状腺主质退行性变,引起滤泡结构的破坏,大量的甲状腺激素和碘比蛋白突然释放入血循环中。甲状腺细胞的受损进一步导致碘的浓缩过程和激素合成的暂时中断。原先合成好的甲状腺内之激素突然释放引起临床上的甲状腺机能亢进,继而因甲状腺合成中断又逐渐出现甲状腺机能不足的病征。本文对2例亚急性甲状腺炎的患者在临床发病后的几周内,对甲状腺代谢的各种指标按时间顺序进行了研究,其目的是观察甲状腺机能异常的程度和本质。发现在疾病早期血清甲状腺素,蛋白结合碘和T_3
Previous studies suggested that the main feature of sub-acute thyroiditis (De Quervain’s thyroiditis) is the degeneration of the thyroid gland, causing the destruction of the follicular structure, and the massive release of thyroid hormones and iodine specific proteins into the bloodstream. Damage to thyroid cells further results in temporary disruption of the iodine concentration process and hormone synthesis. The original synthesis of a good hormone within the thyroid suddenly caused by clinical hyperthyroidism, followed by thyroid synthesis and then gradually interrupted the symptoms of hypothyroidism. In this paper, two cases of subacute thyroiditis in patients within a few weeks after the onset of the clinical parameters of thyroid metabolism were studied in chronological order, its purpose is to observe the extent and nature of thyroid dysfunction. Serum thyroxine, protein-bound iodine and T_3 were found early in the disease