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本文测定了52例正常人,44例病区对照,90例地甲肿病人,33例克汀病人和22例甲亢病人的血清 FT_3、FT_4、TSH 和 Tg。病区各组与甲亢组 Tg 均显著高于正常组;地甲肿和克汀病组 Tg 高于病区对照组。甲状腺Ⅱ°肿大以上者血清 Tg 明显高于甲状腺Ⅰ°肿大者。各组 Tg 和测定激素间未表观相关。结节型地甲肿组血清 Tg 和 FT_3/FT_4比值间表现显著相关;但其他组未发现相关。结果提示地甲肿病人 Tg 升高的原因可能和碘缺乏引起低碘 Tg 合成和周转加速有关。
This article measured the serum FT_3, FT_4, TSH and Tg of 52 normal subjects, 44 cases of ward control, 90 cases of goiter, 33 cases of cretinism and 22 cases of hyperthyroidism. Tg in each group and hyperthyroidism group were significantly higher than those in normal group. Tg in patients with local gingival and cretinism was higher than that in control group. Thyroid enlargement of more than Ⅱ ° serum Tg was significantly higher than those of thyroid Ⅰ ° enlargement. There was no apparent correlation between Tg and hormone in each group. There was a significant correlation between serum Tg and FT_3 / FT_4 ratio in nodular goiter group, but no correlation was found in the other groups. The results suggest that the cause of elevated Tg may be related to iodine deficiency caused by low-iodine Tg synthesis and turnover accelerated.