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心室起搏(VP)引起自身心律改变的报道很多,其发生率为55%～93%,但其机理和意义尚不甚清楚。本文采用体表电位与标测(BSM),并与~(201)铊心肌显像(TⅠ-SC)结果对照,对10例各种原因的缓慢性心律失常患者VP前、中、后进行观察,以探讨上述T波改变的机理。方法:VP和对照组各10例。除外了与VP无关的可能新出现的复极异常,如缺血性心脏病、LBBB和心室肥厚等。VP组除外了按需型起搏时出现窦律的病人。植入永久型双腔起抟器8例,心室起会搏器2例,至本研究开始时已14天至3年。BSM:采用VCM-3000型BSM仪,用87个导联连续在VP前、中和后标测。TI-SC:静息情况下分别在起抟前(3例)、中(8例)和后(3例)进行。并对2例DDD起搏
Ventricular pacing (VP) caused many changes in their own heart rhythm, the incidence rate of 55% to 93%, but the mechanism and significance is not yet clear. In this paper, the body surface potential and mapping (BSM), and with ~ (201) thallium myocardial imaging (T Ⅰ-SC) results compared to 10 patients with various causes of arrhythmia in patients with VP before, during and after observation To explore the mechanism of T-wave changes. Methods: VP and control group, 10 cases each. Excluding VP-related potential new complex abnormalities such as ischemic heart disease, LBBB and ventricular hypertrophy and so on. Patients in the VP group excluded sinus rhythm on demand pacing. 8 cases were implanted with permanent dual chamber cuffs and 2 cases with pacemaker in the ventricle. From the beginning of this study, 14 days to 3 years. BSM: using VCM-3000 BSM instrument, with 87 leads in the VP before and after the neutralization mapping. TI-SC: Resting cases were performed before (3 cases), middle (8 cases) and after (3 cases) respectively. And 2 cases of DDD pacing