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1928年Bagley首次报告了蛛网膜下腔出血后脑积水,近年来,医学文献报导日益增多,已证明,引起慢性交通性或阻塞性脑积水的原因是软脑膜纤维化及细胞、纤维性渗出物阻塞了基底池、第四脑室的孔道和蛛网膜颗粒.一些研究者估计、动脉瘤破裂引起蛛网膜出血后脑积水的发病率为10-19%,如将只有轻度侧脑室扩大包括在内,其发病率稍高些.作者将1968～1969年间肯定或怀疑颅内动脉瘤或动静脉畸形引起蛛网膜下腔出血的48例病人的分析结果与1974～1975年间56例类似的病人进行对比.未用EACA的1968～1969组中,经造影证实为脑积水且有症状者占10%,侧脑室扩大的发病率为17%;应用EACA的1974～75组中,因脑积水而产生症状者占31%,脑室扩大发病率为43%.
Bagley was the first to report hydrocephalus after subarachnoid hemorrhage in 1928. In recent years there has been a growing body of medical literature that has proved that the causes of chronic traffic or obstructive hydrocephalus are pia mater fibrosis and fibrosis Ocular obstruction of the basal cistern, the fourth ventricle and arachnoid granules Some researchers estimate that the rupture of the aneurysm causes the incidence of hydrocephalus after arachnoid hemorrhage is 10-19%, as will be only mild lateral ventricle expansion including , The incidence is slightly higher.The author of the 1968 ~ 1969 confirmed or suspected intracranial aneurysm or arteriovenous malformations caused by subarachnoid hemorrhage in 48 patients with the analysis of results from 1974 to 1975, 56 cases of similar patients Compared with the EACA 1968 ~ 1969 group, confirmed by the hydrothorax and symptoms of symptomatic accounted for 10%, the incidence of lateral ventricle expansion was 17%; using EACA 1974 ~ 75 group, due to the brain volume Water and the symptoms accounted for 31%, the incidence of ventricular enlargement was 43%.