论文部分内容阅读
高浓度吸氧可引起氧中毒远于1878年已由Bert氏首先指出。近30多年来,用氧治疗新生儿窒息及呼吸窘迫症可引起红细胞及视网膜损害(晶体后纤维增生症)亦引起普遍的重视。近些年,国外对氧疗法可引起肺脏损害的报告较前大为增加。因为氧吸入后,呼吸系统为首当其冲的损害部位。Northway于1967年复习文献发现,早产儿给高浓度氧吸入治疗呼吸窘迫症引起支气管肺发育不良(BPD)约占5～68%。现特介绍氧中毒的发病机理及防治于下。
High concentrations of oxygen can cause oxygen poisoning as early as 1878 by Bert’s first pointed out. In the past 30 years, oxygen therapy for neonatal asphyxia and respiratory distress syndrome can cause erythrocyte and retinal damage (posterior fibroplasia syndrome) has also caused widespread attention. In recent years, foreign reports of lung damage caused by oxygen therapy have greatly increased. Because oxygen inhalation, the respiratory system is the first to damage the site. Northway’s review of the literature in 1967 found that premature infants treated with high concentrations of oxygen for respiratory distress caused about 5 to 68% bronchopulmonary dysplasia (BPD). Now introduce the pathogenesis of oxygen poisoning and prevention in the next.