论文部分内容阅读
长期以来，人们研究表明癌是由长久积累的破坏正常细胞生长调控机制的多重变异所引发，不同类型的癌可能都在同一基因上发生变异。近来研究提示：端粒酶的激活机制可能在恶性肿瘤细胞中发挥重要作用，可能是细胞永生的必要条件或者可使细胞无限增生。故此，本文就端粒及端粒酶
For a long time, people have shown that cancer is caused by multiple mutations that have accumulated long-term mechanisms that disrupt normal cell growth. Different types of cancer may mutate in the same gene. Recent studies suggest that the activation mechanism of telomerase may play an important role in malignant cells. It may be a necessary condition for the immortality of cells or allow cells to grow indefinitely. Therefore, this article focuses on telomere and telomerase.