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后适应和胰岛素通过激活磷脂酰肌醇3激酶(phosphatidylinositol-3kinase,PI3K)信号通路发挥心脏保护作用。由于压力超负荷加剧缺血再灌注(ischemia-reperfusion,IR)损伤,该研究为了验证“压力超负荷能够削减后适应和胰岛素介导的心肌保护,这种作用是由于磷脂酰肌醇3激酶-蛋白激
Post-adaptation and insulin exert cardioprotective effects by activating the phosphatidylinositol-3 kinase (PI3K) signaling pathway. Because pressure overload exacerbates ischemia-reperfusion (IR) injury, the study was designed to demonstrate that "pressure overload can reduce post-adaptation and insulin-mediated myocardial protection due to phosphatidylinositol 3 kinase- Protein shock