重症急性钡中毒临床上少见,本院收治3例,兹报道如下:例:女性,37岁,于1984年9月16日上午5时30分服毒,即被送本院。神清,因毒物性质不明,作一般服毒处理,留急诊室观察。上午10时,出现心律失常,心电图示频发室早及阵发性反复短串室速,心肌损害,U 波明显,给利多卡因静滴。下午3时30分,出现四肢瘫痪,昏迷,经调查证实毒物为碳酸钡收治住院。体检:T36℃。P70次/分,R20次/分,BP0。深昏迷,脉细,唇绀,两肺正常呼吸音。心律不规则,四肢软瘫,深浅反射消失。血钾3.0mEq/L。给予吸氧,扩容,升压,补钾,利多卡因静滴及二巯基丁二酸钠静注。终因发现中毒病因较迟,抢救无效,于次日上午3时50分死亡。例2:男性,4岁,因误吃氯化钡作发酵剂做成的馒头3只,出现呕吐,腹泻6小时,进行性呼吸困难伴四肢 Severe acute barium poisoning clinically rare, admitted to our hospital in 3 cases, it is reported as follows: Example: Female, 37 years old, at 05:30 on September 16, 1984 was poisoned, that is, sent to the hospital. God clear, because of the nature of the unknown drugs for general poisoning treatment, emergency room observation. 10 am, arrhythmia, ECG showed frequent premature ventricular paroxysmal and paroxysmal repeated VSV, myocardial damage, U wave significantly to lidocaine intravenously. At 3:30 pm, there were paralyzed limbs and a coma. Investigation confirmed that the poison was admitted to hospital for barium carbonate. Physical examination: T36 ℃. P70 beats / min, R20 beats / min, BP0. Deep coma, pulse thin, cyanotic cyanosis, two lungs normal breath sounds. Irregular heart rhythm, limbs soft paralysis, depth reflex disappeared. Potassium potassium 3.0mEq / L. Give oxygen, expansion, boost, potassium, intravenous injection of lidocaine and sodium dimercaptosuccinate. The end result of the discovery of the cause of poisoning later, the rescue is invalid, died at 3:50 on the next day. Example 2: Male, 4 years old, due to mistaken eating barium chloride as a starter 3 made bread, vomiting, diarrhea 6 hours, progressive dyspareunia with limbs