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作者从三个不同层次(整体肺、离体肺及肺血管内皮细胞)的肺损伤模型证实了油酸引起的急性肺水肿中有氧自由基参与,来自嘌呤氧化酶的氧自由基起着原发始动作用,而来自嗜中性白细胞的氧自由基起着继发和扩大损伤的作用。
The authors demonstrated the involvement of oxygen free radicals in acute pulmonary edema induced by oleic acid from lung injury models at three different levels (whole lung, isolated lung, and pulmonary vascular endothelial cells). The oxygen free radicals from purine oxidase act as original Start with the action, and from the neutrophil oxygen free radicals play a secondary and expand the role of injury.