患者男性,74岁.因排尿不畅5天、尿潴留3天,于1990年5月28日入院。检查:心肺正常,浅表淋巴结不肿大。肛指检查:前列腺Ⅱ°增大、无结节。B超示前列腺增生,左肾盂积水伴输尿管上段扩张。静脉肾盂造影:左肾未显影。膀镜镜检查:前列腺中度增大。左肾盂逆行造影见输尿管中段有一长约2cm的狭窄段。同年6月14日手术探查,见左髂总动脉分叉处下方1cm处输尿管管壁增厚,僵硬和周围组织粘连,上端输尿管扩张。探查膀胱未见占位病变。行输尿管部分切除端端吻合及前列腺摘除术。标本镜下观:前列腺的大部分结构被破坏,癌细胞部分形成腺管,部分呈筛状、条索状或散在分布在间质内,个别腺腔内可见嗜酸性分泌物或由分泌物聚成的淀粉样小体。输尿管移行上皮正 Male patient, aged 74. Due to poor urination 5 days, urinary retention for 3 days, on May 28, 1990 admission. Check: normal cardiopulmonary, superficial lymph nodes are not swollen. Anal finger examination: Prostate Ⅱ ° increase, no nodules. B ultrasound shows benign prostatic hyperplasia, left hydronephrosis with ureteral dilatation. Intravenous pyelography: left kidney not developed. Mirroroscopy: moderately enlarged prostate. Left renal pelvis retrograde angiography see the middle of the ureter has a length of about 2cm of the narrow segment. June 14 the same year surgical exploration, see the left common iliac artery bifurcation 1cm below the ureteral wall thickening, stiffness and the surrounding tissue adhesions, the upper ureteral dilatation. Exploring the bladder no space lesions. Partial ureter resection end anastomosis and prostatectomy. Specimen microscopic view: most of the prostate structure is destroyed, part of the cancer cells to form duct, part of the mesh-like, cord-like or interspersed within the interstitial, some glandular eosinophilic secretions can be seen or secreted by the poly Into the amyloid body. Ureteral transitional epithelium is positive