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A20 was originally characterized as a TNF-inducible gene in human umbilical vein endothelial cells. As an inhibitor of NF-kappaB signaling, A20 could protect apoptosis, inflammatory and cardiac hypertrophy. We investigated the role of A20 on acute myoca
A20 was originally characterized as a TNF-inducible gene in human umbilical vein endothelial cells. As an inhibitor of NF-kappaB signaling, A20 could protect apoptosis, inflammatory and cardiac hypertrophy. We investigated the role of A20 on acute myoca