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在硫贲妥钠麻醉的狗中,对椎动脉内压减低时血压变化的机理进行了分析和探讨。在夹闭双侧颈总动脉后,阻断椎动脉血流,可引起动脉血压升高。但在夹闭双侧颈总动脉前,则不能引起这种反应。在双侧颈动脉窦神经被切断或颈内、外动脉被结扎的动物中,夹闭双侧颈总动脉后阻断椎动脉血流,也不能引起动脉血压升高。这些结果表明:在夹闭双侧颈总动脉后,椎动脉内压减低时的血压升高,不是通过椎动脉内感受器的作用,而是通过颈动脉窦区的反射机制引起的。
The mechanism of the change of blood pressure when vertebral artery pressure was reduced was analyzed and discussed in the rat anesthetized with thiopental sodium. Clipping the bilateral common carotid artery, blocking vertebral artery blood flow, can cause arterial blood pressure. However, in clamping bilateral common carotid artery, it can not cause this response. In the bilateral carotid sinus nerve was cut or the internal and external arteries were ligation of animals, the bilateral common carotid artery occlusion blocked vertebral artery blood flow, it can not cause arterial blood pressure. These results indicate that the elevation of blood pressure during the reduction of the internal pressure of the vertebral artery after occlusion of the bilateral common carotid arteries is caused not by the action of the vertebral artery receptors but by the reflex mechanism of the carotid sinus.