肝硬化门脉高压时由于外周动脉扩张 ,造成动脉血管腔隙充盈不足 ,激活体内交感神经和血管活性物质 ,试图来代偿有效循环血容量的不足 ,结果造成肾脏低灌注 ,引起钠水潴留 ,参与肝硬化门脉高压高动力循环状态和腹水的形成 ,当此种代偿性机制衰败后 ,则形成顽固性腹水 ,甚至出现 Cirrhosis due to portal hypertension due to peripheral arterial dilatation, resulting in inadequate filling of the arterial lacuna, activating the body’s sympathetic and vasoactive substances in an attempt to compensate for the lack of effective circulating blood volume, resulting in low renal perfusion, causing sodium and water retention, Participate in the cirrhosis of the portal hypertension hyperdynamic circulation and the formation of ascites, when the compensatory mechanism of decay, the formation of intractable ascites, or even