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黄瞟吟氧化酶（EC1．2．3．1XOD）广泛分布于体内各组织中，心、肾、小肠缺血再灌注时在XOD作用下产生大量氧自由基，造成组织损伤。用链脲佐菌素（STZ）诱导的迟发性糖尿病大鼠，体内自由基防御机制受损是β细胞损害的先决条件与决定因素[1]，XOD在体内有无改变，本实验通过检
Huangqi Yin oxidase (EC1.2.3.1XOD) are widely distributed in the body of various tissues, heart, kidney, small intestine during ischemia-reperfusion produced a large number of oxygen free radicals under the action of XOD, resulting in tissue damage. STZ-induced delayed-onset diabetes mellitus in rats with impaired free radical defense is a prerequisite and determinant of β-cell damage [1], whether XOD has been altered in vivo,